​​I have been researching Cyplexinol for a while and now with additional research I am comfortable mentioning it to you.

Platelet Rich Plasma Therapy  stimulates Growth Factors in high concentrations.  it triggers a recruits stem cell activation.

4-12 months is the recommended time to accrue bone rebuilding results

  • Some Bone Facts:
  • Bones loss is primarily caused by decrease in Estrogen
  • It takes years  / lifetime to develop
  • It has nothing to do with what you eat and all to do with what we ABSORB
  • Bone health has little to do with Bone Density

I still have faith in Magnifical ; Vit. D;  Magnesium (Re-Mag) and a touch of Calcium (Calphonite) ; Plant based Xylitol used in Europe;as well as Progesterone or DHEA to help regulate Estrogen.

Overall and in general I am not a fan of  Bisphosphonates  with exception to  HR positive HER 2 negative individuals using estrogen blockers as part of their treatment protocol.

And I must admit, none of my patients have ever had an outright bone fracture as  result of Osteoporosis.However, Platelet Rich Plasma, Stem Cell Growth and what triggers stem cell growth may be the next frontier in keeping bones healthy.. 

Please read a little bit on Cyplexinol and as always know that any product I recommend is the best on the planet. This applies to Cyplexinol and everything else originating from this office.

AND please pass this info onto others who are experiencing either Osteopenia and or Osteoporosis.For those who cannot make it in to  see me or  live in “my neighborhood”, I offer Zoom consultations


Stem Cell and Research Therapy—-Cyplexinol

Cyplexinol® is an all-natural stem cell signaling protein complex[,] which uniquely stimulates key pathways to help support your body’s ability to create new bone and cartilage tissue!

Cyplexinol®  is a natural BMP complex, which consists of a collagen fragment matrix with the BMPs and endogenous growth factors bound within and to the matrix.

Bone morphogenetic proteins (BMPs), originally identified as osteoinductive components in extracts derived from bone, are now known to play important roles in a wide array of processes during formation and maintenance of various organs including bone, cartilage, muscle, kidney, and blood vessels.

Contact me for further information!

To your health,

Fermented Greens – The Key to Unlocking Complete Nutrient Vitality

Fermented Greens – The Key to Unlocking Complete Nutrient Vitality

Green smoothie with spirulina,spinach and blueberries

Acid Alkaline Balance and Its Impact on Health

Acid alkaline balance is a crucial factor in our overall health. For our cells to function properly, the balance between acidic and alkaline substances in the body must be tightly regulated so that the pH of the blood is slightly alkaline, around 7.4. When our bodies become too acidic, known as acidosis, we may experience decreased immune function, chronic inflammation, and even bone loss. Conversely, an overly alkaline body, known as alkalosis, can cause muscle weakness and cramping.

To achieve a healthy acid alkaline balance, our diet plays a vital role. Consuming alkalizing minerals such as potassium, calcium, and magnesium found in greens can help balance the acid levels in the body. Greens like spinach, kale, and collard greens are rich in alkalizing minerals and vitamins that promote a healthy pH balance. Additionally, herbs such as parsley and cilantro are great sources of alkalizing minerals.

A QRA Perspective on pH

But the benefits of maintaining a healthy acid alkaline balance extend beyond just physical health. Energetically, an alkaline body promotes vitality, better digestion, and overall wellness. It’s no wonder why many health experts consider pH balance as one of the essential components of optimal health.

As Quantum Reflex Analysis (QRA) practitioners, we believe that the body’s energy systems and organs can become imbalanced when the acid-alkaline balance is disrupted. By using muscle testing to identify these imbalances we can recommend natural remedies, such as whole food supplements, homeopathy, and herbal formulas, to help correct them. By incorporating QRA concepts into the discussion of acid-alkaline balance, we can consider not just the physical effects of maintaining a healthy pH balance, but also the energetic benefits.

By ensuring that our body is properly balanced and nourished with alkalizing minerals, we can promote vitality, better digestion, and overall wellness. Including alkalizing greens and herbs in our diet can help us achieve a healthy acid-alkaline balance, and incorporating QRA concepts may help us identify and correct any imbalances that may arise.

It All Begins with Soil Health …

As discussed in our recent Cal-Mag blog, the pH of the soil has considerable influence on the mineral content of food. One of the first considerations in an alkaline diet, which includes an emphasis on consuming more fruits and vegetables, is to know what type of soil they were grown in, and whether done so using organic or convention farming practices. Since the agricultural revolution and even more recently with industrialized food production, there has been a decrease in potassium compared to sodium and an increase in chloride compared to bicarbonate found in the diet.

You see, minerals are used as buffers to maintain pH. In the soil, when the pH is less than 6, there is less calcium and magnesium while in soils with a more alkaline pH causes iron, manganese, copper and zinc to become chemically unavailable. The pH acts as the gatekeeper and only allows minerals to be utilized and absorbed when the pH is within a specific range.

This scenario plays out in the human body as well. An acidic pH can hamper the bioavailability of nutrients for uptake by our cells. Blood has a very narrow pH range (7.35 – 7.45) which is tightly regulated by our respiratory and urinary systems to achieve acid-base in the body. The lungs help regulate blood pH by removing carbon dioxide from the blood through the lungs, where it is exhaled. With the help of the brain, the speed at which carbon dioxide is exhaled is regulated, minute by minute. In a process that occurs over several days, the kidneys also help regulate blood pH by excreting excess acids or bases metabolized from the diet.

Just like adding alkalinizing soil amendments, such as dolomite lime, when individuals begin to consume more alkaline foods, the intracellular terrain slowly begins to change, improving the exchange of nutrients and waste at the cellular level. When consistently consuming more alkalizing fruits and vegetables, significant pH changes may be detectable via urine or saliva, but not in blood pH.

Acid-Base Disorders – What are They and Why Worry?

Alkalosis and acidosis are disorders that result from complications maintaining acid-base balance. Alkalosis occurs when blood pH increases, becoming less acidic while acidosis occurs when blood pH decreases, becoming more acidic.

Metabolic acidosis and metabolic alkalosis are caused by an imbalance in the production of acids or bases as well as their excretion by the kidneys. This occurs when the body is in shock or when type 1 diabetes is poorly controlled resulting in diabetic ketoacidosis or worse, kidney failure. Respiratory acidosis and respiratory alkalosis are caused by changes in the carbon dioxide exhalation due to lung or breathing disorders such as pneumonia, asthma, COPD/sleep apnea, or heart failure. When either of these disorders is detected, it provides an important clue to doctors that a serious problem may be brewing in the body.

When our bodies are in a more acidic state, they are weaker and more vulnerable to disease and dysfunction. Our body’s natural defenses and ability to repair from wear and tear become weakened. Conditions such as chronic renal failure and chronic metabolic acidosis inevitably result in a loss of skeletal muscle.

Chronic Acidosis – A Recipe for Trouble 

Metabolic acidosis can be directly associated with consumption of modern, SAD diets. Due to the cumulative effects of soil depletion, modern human diets are deficient in magnesium, potassium, as well as fiber and contain excessive amounts of saturated fat, simple sugars, sodium, and chloride. Over time, this sets the stage for metabolic acidosis to occur due to the gradual loss of the kidney’s acid-base regulatory function which may have adverse effects on bone health as well as body systems and organs, especially the kidneys.

Studies show that even the slightest shift toward metabolic acidosis can produce insulin resistance and systemic hypertension. Furthermore, strongly acidic diets combined with excess body weight, lack of exercise and ageing, are a recipe for metabolic syndrome and type 2 diabetes.

Dietary sodium is a predictor increased incidence of metabolic acidosis. When our bodies become too acidic, calcium and magnesium are drawn from the bones. Bone building cells become less effective, and the cells’ pH balance is affected. This condition, called chronic metabolic acidosis, can deplete the mineral reserves in bone and can cause osteopenia (lower bone density) and eventually osteoporosis (loss of bone mass with risk of fractures).

The kidneys, one of the most vital organ systems of the body, can become overwhelmed by net acid loads resulting in higher incidence of kidney disorders including kidney stones, kidney disease, and kidney failure. A low carb, high protein diet with its increased acid load results in very little change in blood pH, but results in many observable changes in urinary chemistry. Urinary magnesium levels, urinary citrate and pH are decreased, while urinary calcium, undissociated uric acid, and phosphate are increased in individuals consuming modern, SAD diets.

Greens – A Vital Solution for Maintaining Acid Alkaline Balance

As wellness practitioners, we already know that greens are a powerful solution to maintaining a healthy acid alkaline balance in the body. These nutrient-dense plants contain alkalizing minerals, vitamins, and antioxidants that help to regulate pH levels and promote optimal health. For example, kale is an excellent source of calcium, spinach is rich in magnesium, and collard greens are loaded with potassium, all of which help to neutralize acidity and promote a healthy pH balance.

But the benefits of greens don’t stop there. They are also incredibly nutrient-dense and offer a range of health benefits. For instance, greens like arugula, watercress, and chard are rich in antioxidants that help to protect the body against oxidative stress and inflammation. They also contain a wide range of vitamins and minerals that are essential for maintaining optimal health.

Alkaline diets, or diets high in fruits and vegetables, improve the potassium-sodium ratio improving cardiovascular health, bone health while reducing muscle loss and mitigating chronic diseases such as hypertension and strokes. The increase in dietary magnesium, required to activate vitamin D, would enhance bone health as well as immune function.

Energetically, whole foods, especially iron-containing greens, build blood and improve energy levels by enhancing RBC production. They also support healthy digestion, which is essential for optimal nutrient absorption. Adding greens to your diet is an easy way to boost your overall health and energy levels.

For those who struggle to consume enough greens in their daily diets, powdered greens supplements can be a convenient solution. Look for supplements that have been fermented, as fermentation can enhance nutrient absorption and improve the bioavailability of the nutrients in the greens.

Fermentation – Traditional Methods for Optimizing Nutrition

Fermentation has been used for centuries to enhance the nutritional value of foods. When it comes to leafy green herbs, vegetables and seaweeds, fermentation can improve nutrient absorption and enhance their bioavailability, making it easier for the body to access and utilize their beneficial nutrients. This ancient technique of preparing and preserving vegetables and fruits has several confirmed benefits including enhanced digestibility of macronutrients, removal of anti-nutritional factors (such as phytic acid, oxalates, and tannins), and promotes beneficial gut bacteria to produce probiotics, enzymes, and organic acids that promote healthy digestion and boost immune function.

Studies have shown that fermenting greens increases the bioavailability of nutrients such as vitamins C, E, and K, calcium, and iron. This means that consuming fermented greens supplements can provide even more nutritional benefits than fresh or cooked greens. Additionally, fermented greens supplements can be more convenient and easier to consume than whole greens for individuals with exceptionally busy lives or an exceptional nutritional need.

Choosing a high-quality fermented greens supplement made from organic, non-GMO ingredients free from harmful fillers, or additives is crucial. Fermented greens supplements can boost your intake of greens, enhance nutrient absorption, and promote a healthy acid alkaline balance. But fermentation isn’t just about nutrition. Consuming fermented foods enhances vitality and digestion, making it easier for the body to build blood and energy levels. So, if you’re looking to improve your acid alkaline balance, enhance nutrient absorption, and boost your vitality, give fermented greens a try.

Assessing Resonance of Patients and Plants

As natural health professionals, we can use QRA to determine which powdered greens supplements are the best fit for each patient’s unique needs. Through non-invasive testing of the body’s reflexes, we can identify which supplements are most effective at promoting optimal nutrient absorption and utilization for each individual.

In addition, QRA can help us to determine the ideal serving size of powdered greens supplements for each patient. By testing the body’s reflexes, we can identify the most effective dosage to support the body’s needs.

Furthermore, QRA can help us to identify any potential allergies or sensitivities to specific powdered greens supplements, ensuring that our patients are only consuming supplements that are safe and beneficial for their unique body.

By incorporating QRA into our approach to powdered greens supplements, we can provide our patients with personalized recommendations that support their overall health and well-being. We can empower our patients to take charge of their health by offering practical solutions that fit their lifestyle, while still emphasizing the importance of a healthy diet as the foundation of optimal health.

Powered by Plants – The Practicality of Using Powdered Greens Supplements

Our role as wellness practitioners, is to empower our patients to take charge of their health. And while fresh greens are always the best option, we understand that life can get busy. That’s where powdered greens supplements come in – they offer an easy and convenient way to get the nutrients your body needs.

But as with any supplement, quality is key. Look for organic, non-GMO, and a fermented greens options to ensure optimal nutrient absorption. Powdered greens supplements are also more concentrated than fresh greens, meaning you can pack more nutrients into a smaller serving, making this dietary supplement practical and potent!

Another benefit of powdered greens supplements is their longer shelf life. They can be a great option for patients with busy lifestyles who struggle to eat fresh greens regularly. However, it’s important to note that they should not be a substitute for a healthy diet. Instead, encourage your patients to use them as a supplement to their daily greens.

As natural health professionals, it’s our duty to educate our patients on the importance of a healthy diet and offer solutions that fit their lifestyle. Utilizing high-quality powdered fermented greens supplements can be a practical solution for patients who need an easy and convenient way to get their daily dose of greens. With the right supplement, our patients can boost their nutrient intake and energy levels without sacrificing time and convenience.


Jaffe, Russell. Joy in Living: The Alkaline Way. Health Studies Collegium. 2014.

Schwalfenberg, Gerry. The Alklaine Diet: Is There Evidence That an Alkaline pH Diet Benefits Health? Journal of Environmental and Public Health. Volume 2012, Article ID 727630, 7 pages doi:10.1155/2012/72763

Shalchi, Homa. Debating Diets: What is the alkaline diet? February 28, 2022. Baylor College of Medicine. Accessed at   

Lewis III, James L. Overview of Acid-Base Balance. Merck Manual Consumer Version. Available at:  

Ray, Ramesh, and Swain M. Chapter 3 – Nutritional Values and Bioactive Compounds in Lactic Acid Fermented Vegetables and Fruits. Lactic Acid Fermentation of Fruits and Vegetables. 2017.

Question: What is the importance of maintaining an acid-alkaline balance?

Answer: Maintaining an acid-alkaline balance is essential for our cells to function properly. An overly acidic or alkaline body can cause decreased immune function, chronic inflammation, and even bone loss.

Question: What foods can help promote a healthy pH balance?

Answer: Greens such as spinach, kale, and collard greens are rich in alkalizing minerals and vitamins that promote a healthy pH balance. Additionally, herbs such as parsley and cilantro are great sources of alkalizing minerals.

Question: What are the benefits of consuming fermented greens?

Answer: Consuming fermented greens can improve nutrient absorption and enhance their bioavailability, making it easier for the body to access and utilize their beneficial nutrients. It can also increase the bioavailability of vitamins C, E, and K, calcium, and iron, and promote beneficial gut bacteria to produce probiotics, enzymes, and organic acids that promote healthy digestion and boost immune function. 



  • Your teeth are organs that are connected to your nervous system via the trigeminal nerve, one of the 12 cranial nerves
  • In his book, “It’s All in Your Mouth: Biological Dentistry and the Surprising Impact of Oral Health on Whole Body Wellness,” biological dentist Dr. Dominik Nischwitz presents a holistic treatment program for whole-body health and well-being
  • Nischwitz has developed a Bone Healing Protocol that includes micronutrients such as vitamins D, C and K2, magnesium and B vitamins. Your vitamin D3 level should ideally be above 60 ng/mL for proper bone formation
  • His holistic treatment plan includes removing or addressing oral interferences such as metal fillings, root canals and cavitations
  • Cavities and resulting problems can be avoided altogether by following an optimized lifestyle program where you’re getting ideal levels of sun exposure and nutrition, and avoid toxins and certain dietary culprits such as gluten and dairy

In this interview, Dr. Dominik Nischwitz, author of “It’s All in Your Mouth: Biological Dentistry and the Surprising Impact of Oral Health on Whole Body Wellness,” discusses his “all-in-one” holistic treatment program, and why addressing your oral health can have far-reaching whole body benefits.

Nischwitz is a biological dentist, like his father before him, and has served as president of the International Society of Metal Free Implantology (ISMI) since 2019.

As a child, Nischwitz “was always a little bit sick” and was frequently treated with antibiotics for recurrent throat infections. At the age of 14, his wisdom teeth were extracted. Age 15 brought on appendicitis and severe acne, which were also treated with antibiotics for months at a time.

“I thought it was normal to be just a little sick and then a little healthy … Then they wanted to take out my tonsils when I was 16. My mom said, ‘Ah, let’s get a second opinion. Go to this naturopathic doctor’ … He tested me with kinesiology and … told me, ‘You’re just allergic to milk … This is an allergy.’

[Removing milk] helped. My tonsils are still in there and it took me a few more years to actually look into dentistry. I was [doing] civil service at a Red Cross as a paramedic … and you have to do an internship in the clinic.

Maybe it’s coincidence. They put me in dental clinic … I applied to university, made the cut, and started without knowing anything. Finally, I got interested [in health] … because at the same time, I was starting to work out. I just wanted to perform better.”

Following the advice given in a bodybuilding magazine, Nischwitz started eating 3,000 calories a day to gain mass. Little did he know all calories are not equal, and after a year of eating noodles and tuna, he’d gained 20 kilos (about 44 pounds), although it wasn’t all muscle.

“I didn’t know it was health that I was missing. I was just focusing on performance and of course muscle gain. Maybe it was just an aesthetic thing, but I learned everything I could about nutrition …

I tried every possible diet. I used every supplement. I was the guy in university who had his box of food always with him, not even knowing that it maybe was also too much carbs … But I learned a lot from this.”

Eventually, Nischwitz learned about the effects dental amalgam has on health, which “totally clicked with everything nutrition-wise” he was learning. “Every minute in my residency when I wasn’t drilling out amalgam fillings, I was searching the internet for stuff to remove, to detox, how the liver phases work, basically, everything possible,” he says. “I was really curious to find real solutions.”

The All-in-One Concept of Health

Nischwitz now focuses on what he calls an “all-in-one concept” of health, starting with the mouth.

“Your teeth are organs that are connected to your whole nervous system and basically are part of your brain, kind of like your eyes,” he says. “You have this massive brain nerve there called the trigeminal nerve. It’s one of the 12 cranial nerves and takes up 50% of the space of all the other ones, so it’s quite important.”

To get started, patients will send him their current panoramic X-rays, a medical questionnaire, and their vitamin D3 and LDL blood work to provide some basic knowledge of their health.

Next, the patient must be properly prepared through nutrition and other lifestyle changes. “It’s basically the same stuff you would do for a patient of every other functional medicine doctor. Go as natural as possible,” Nischwitz says.

His book contains two charts, one red and one green. The red chart describes common food intolerances and food toxins, and their alternatives. All patients are asked to go grain- and dairy-free. The green chart lists healthy carbohydrates, healthy fats and proteins to add to your diet.

Nischwitz has also developed a Bone Healing Protocol that includes certain micronutrients and focuses on high doses of vitamin D3. Vitamin D levels, for example, should ideally be above 60 nanograms per milliliter (ng/mL), and if your levels are low to begin with, you may need to take a supplement for several months before your treatments begin. The protocol also stresses magnesium, vitamin K2, vitamin C and B vitamins.

“So, they come into this health optimization week already immunologically boosted,” Nischwitz. “The nervous system is already very good. What we’re trying to achieve then is bring the patient from chronic sympathetic nervous system mode into parasympathetic [mode] …”

Removing Oral Factors That Interfere With Health

Nischwitz’s all-in-one treatment plan includes removing or addressing oral interference such as:

Metal fillings — Mercury (amalgam) fillings (and all other metal restorations) are removed safely using specialized rubber dams and cleanup suction, as well as other parameters to ensure mercury vapors are not recirculated into the system, as this can lead to acute toxic overload. Patients will also receive a nasal probe and intravenous nutrients to assist with detoxification and healing.

Root canals (replacing them with ceramic implants) — Root canaled teeth are extracted and the socket cleaned with ozone. Neural therapy is also used, along with platelet-rich fibrin (APRF) treatment.

“We draw blood before the surgery and spin it in the centrifuge to make APRF,” he explains. APRF contains stem cells and growth factors that are then placed into the empty socket or beside the zirconium dioxide implant, which has no metal oxides in it and is completely biocompatible.

On a side note, while few dentists are educated about this, there are ways to rescue an infected tooth, thus avoiding the need for a root canal in the first place. One method that appears to be very effective is sterilizing the infected root with a high-powered YAG laser, which combines both light therapy and acoustic sound therapy, along with ozone.

This has been shown to eliminate the need for a root canal in many cases. What Nischwitz is describing is the necessary rescue effort after a root canal has been performed.

Cavitations — Cavitations is the layman’s term for fatty degenerative osteonecrotic jawbone (FDOJ) or chronic ischemic bone disease (CIBD). It’s also known as neuralgia-inducing-cavitational-osteonecrosis (NICO cavitations).

Cavitations at the back of your jaw left over from wisdom tooth extractions (or every other extraction site) can cause pain to radiate through your other teeth via the trigeminal nerve. Sometimes, patients end up getting a root canal in a painful tooth that really didn’t need it, as the pain actually was caused by a cavitation.

Aside from cavitations, Nischwitz points out that pain in a tooth could also be due to high blood sugar, low blood sugar, an overall change of pH level in your mouth, or a mineral deficiency.

After surgery, Nischwitz prepares a “food design” or “lifestyle concept” plan for each patient, based on body composition, metabolism and so on.

More Information

Nischwitz firmly believes cavities and resulting problems can be avoided altogether by following an optimized lifestyle program where you’re getting ideal levels of sun exposure, nutrition, and avoid toxins and certain dietary culprits such as gluten and dairy.

“This is why it’s so important that all this information comes out,” he says. “And that’s why I devoted a whole chapter to the nutritional part. Basically, biological dentistry, I would say, is an overlap of functional medicine, biohacking and high tech dentistry with the goal of optimal health. I’m a big fan of the basics, and the goal is [changing] the lifestyle …

Everybody talks about leaky gut, but nobody talks about leaky gum. The gum tissue, the gingiva, is the same tissue, it’s squamous tissue. And if you have a chronic gingivitis, for example, just from a lack of nutrients or maybe from the wrong [dental] restorations, you will have an opening into your system because the gingiva is outside.

Your bacteria cannot go really inside [the tooth], but if there’s an opening in the gum or if you have a titanium implant where the tissue doesn’t grow on top, you will always have a huge gap … and all these bacteria, mainly anaerobic bacteria, will travel into your system. This is basically leaky gum. [The problem] starts there, because the gut basically starts in your mouth.”

Nischwitz also stresses the importance of a natural birth (whenever possible) and breastfeeding, as this is really important for proper jaw formation. Breastfeeding requires far greater strength than sucking formula from a bottle, so sucking on the breast develops the lower jaw and muscles, and ensures the proper development of the baby’s palate.

“This is the fulfilling part for me — that I can help you optimize your health. And this is also the challenge that I put onto all my patients; they have to change their lifestyle. Otherwise, we don’t even accept them because then they won’t have good results.”

To learn more, be sure to pick up a copy of “It’s All in Your Mouth: Biological Dentistry and the Surprising Impact of Oral Health on Whole Body Wellness.” You can also find more information about Nischwitz’ practice on his website, DNA Health & Aesthetics. Other resources include his YouTube channel and Instagram.

The Spiritual Consequences of Alcohol Consumption

The Spiritual Consequences of Alcohol Consumption

Although it is mass-produced, mass promoted, legal, and ingested by a multitude of people all over the world, most people don’t ever consider or understand the spiritual consequences of drinking alcohol.

Let’s begin by taking a look at the etymology of the Word alcohol. Etymology means the root of the word… where it is derived from.

The word “Alcohol” comes from the Arabic “al-kuhl” which means “BODY EATING SPIRIT”, and gives root origins to the English term for “ghoul”. In Middle Eastern folklore, a “ghoul” is an evil demon thought to eat human bodies, either as stolen corpses or as children.

The words “alembic” and “alcohol”, both metaphors for aqua vitae or “life water” and “spirit”, often refer to a distilled liquid that came from magical explorations in Middle Eastern alchemy.

In the words of writer and health enthusiast, Jason Christoff :

“In alchemy, alcohol is used to extract the soul essence of an entity. Hence its’ use in extracting essences for essential oils, and the sterilization of medical instruments. By consuming alcohol into the body, it in effect extracts the very essence of the soul, allowing the body to be more susceptible to neighboring entities most of which are of low frequencies (why do you think we call certain alcoholic beverages “SPIRITS?”). That is why people who consume excessive amounts of alcohol often black out, not remembering what happened. This happens when the good soul (we were sent here with) leaves because the living conditions are too polluted and too traumatic to tolerate. The good soul jettisons the body, staying connected to a tether, and a dark entity takes the body for a joy ride around the block, often in a hedonistic and self-serving illogical rampage. Our bodies are cars for spirits. If one leaves, another can take the car for a ride. Essentially when someone goes dark after drinking alcohol or polluting themselves in many other ways, their body often becomes possessed by another entity.”

I became aware of this phenomenon years ago when I was given a spiritual vision. In this vision, I was transported as an observer above a popular bar and nightclub. Above the venue where a variety of ghoul-like entities. Inside the bar were people drinking alcohol, socializing, dancing, and so on. I watched as certain people became very drunk. I saw their souls, while connected through a thread, exited the body. I understood that the soul was leaving the body because of the great discomfort of being in a body highly intoxicated with alcohol. When the soul exited the body, other non-benevolent entities entered or latched on to their vacant shells. Once the entities took hold of the body, they used the body to play out all kinds of dark acts, such as violence, low-level sexual encounters, destructive behaviors, rape, and more.

Years later, while reading a book called Mans Eternal Quest, by Paramahansa Yogananda, this spiritual master clearly explained the exact same thing as I was shown in the vision.

I began to look back over my life and remember situations where I saw dark spirits hanging around people who had become very drunk. Let me elaborate a bit when I say I saw these entities … I have had the abilities of clairvoyance (the ability to perceive things beyond the natural range of the senses … which can include: ESP, extrasensory perception, sixth sense, psychic powers, second sight; telepathy, and more) , clairaudience ( the ability to perceive sounds or words from outside sources in the spirit world), and the experience of being a spiritual intuitive and empath since childhood. I have the ability to see energies and spiritual manifestations that most people don’t see. As I looked back over my life I could remember many incidents of encountering non-benevolent spirits in the presence of intoxicated individuals. I also have had experiences of looking into the eyes of a few people who were surely “possessed” by dark energies that were not their own.

I also remember a psychology course I once took. In part of this course, we studied advertising and the effects on humans. We looked at the advertising for alcohol. A master teacher of this subject illuminated the fact that most alcohol advertisements are embedded with hidden messages and images – not typically perceivable to the common sight, yet perceived through the subconscious. Knowing how powerful the subconscious is in our decision making, feelings, reactions, beliefs, etc., the slick sales teams of alcohol (as well as tobacco and other products) used this sinister technique to trick us into buying their products and joining the societal cult of mental apathy and cultural obedience. Many of these hidden messages and images were extremely sexual – working to influence some of the basest urges and primal nature of humans. Let this example bring you to a place of curiosity and questioning. Why have the marketing teams felt the need to trick us and coerce us through subliminal messages to buy products that are harmful to the human body and to our soul?

How many times have you or someone you know, after becoming quite intoxicated with alcohol, behaved in a manner uncommon to them? Perhaps you experienced the changing of voice, violence, sexual promiscuity, ingesting of harmful substances, destruction to property, conflictual behavior, and other negative expressions. Consider these experiences and ask yourself – is this the manifestation of light, love, and positivity? Do these occurrences represent a path of consciousness and health?

It is a known by many that ingesting alcohol depresses the nervous system, kills brain cells, is toxic to the liver, weakens the immune system, and has many other harmful effects. We are taught that long-term alcohol use can lead to unwanted weight gain, diseases of the liver, lowering of intelligence, and negative effects on hormones. Drinking alcohol while pregnant can lead to birth defects, mental retardation, and deformities in the developing fetus. Yet still, it is mass promoted and supported by our mainstream culture. Have you ever considered that alcohol is a slick tool of the supporters of the Matrix (global mind control and oppression program) to keep people on a path of disempowerment and sickness?

We have to ask why is alcohol legal throughout most of the world, yet in many countries, and specifically the United States, psychedelics are illegal. The conscious and safe use of psychedelics or “visionary medicines”are known to assist in mind expansion, to initiate spiritual experiences where people have communed with the divine, healed numerous physical and spiritual ailments, increase intelligence, help to re-pattern the brain in a positive way, assist people in aligning with their soul’s purpose, and have inspired many people to create great works of art and other innovative creations. It seems that these substances would definitely be banned and discouraged if there truly is an agenda seeking to oppress the human potential and keep us “in the dark” regarding who we are as spiritual beings, our innate potential, and the path to empowerment.

As we strive to heal, awaken, and transform our world – I pray that we wise up to the dirty trick played upon humanity in regards to alcohol. Non-benevolent forces have wanted to keep us oppressed, disempowered, and asleep.

How many of us have seen families broken and lives lost because of alcohol and alcoholism?

Do you think it makes us smarter or healthier or overall better people?

It’s time to change things.

Let’s stand behind replacing the rampant abuse of alcohol with more health enhancing practices and activities – and learn how to live awakened and empowered lives!

Before I close this writing, I want to share a little more about the history of the word alcohol. There have been some people who look into the etymology and discover this explanation –

“alcohol (n.) – 1540s (early 15c. as alcofol), “fine powder produced by sublimation,” from Medieval Latin alcohol “powdered ore of antimony,” from Arabic al-kuhul “kohl,” the fine metallic powder used to darken the eyelids, from kahala “to stain, paint.”

Paracelsus (1493-1541) used the word to refer to a fine powder but also a volatile liquid. By 1670s it was being used in English for “any sublimated substance, the pure spirit of anything,” including liquids. Sense of “intoxicating ingredient in strong liquor” is first recorded 1753, short for alcohol of wine, which was extended to “the intoxicating element in fermented liquors.” In organic chemistry, the word was extended 1850 to the class of compounds of the same type as this.”

Upon further research, we can find that in ancient Egypt, the eyes of both men and women were lined top and bottom with a thick black powder known as kohl, kajal, or mesdemet. The outlined eye resembled the almond-shaped eye of the falcon god Horus observed in the Eye of Horus glyph. It was believed that this shape invoked the god´s protection and warded off evil spirits.

Yet if one were to dig deeper, as a true scientist, researcher, or truth seeker does, you will also discover these interesting facts…

  1. Dr. Rachel Hajar, an accomplished modern-day editor, author and medical advisor, while researching an article on alcohol for her online medical journal, found additional meanings in ancient Arabic texts;
  2. Al kol: Genie or spirit that takes on varied shapes or a supernatural creature in Arabic mythology.
  3. Al kol: Any drug or substance that takes away the mind or covers it.”
  4. The word alcohol is also linked to the fixed star in astronomy known as Algol- also known as “the Demon’s head.”
  5. The current Arabic name for alcohol (ethanol) is الغول al-ġawl – properly meaning “spirit” or “demon”.

It is not a coincidence that alcohol has often been referred to as spirits. There is a deep history behind this intoxicating substance. There are layers of information throughout our culture, sometimes we have to look below the surface of things to find the fullness of truth. I encourage you to deeply consider the information shared here, look at the effects of alcohol in your life, in the lives of the people you know, and in society at large. Make conscious, informed, and health enhancing decisions. The more people who awaken to truth and seek health and liberation from mind control agendas, the more likely we are to make positive changes and co-create a world we feel good about living in.
















San Diego Dates October/December 2022

San Diego Dates October/December 2022

3344 4th Ave.
San Diego 92103
Suite 200 Room 8
Parking Space # 6 behind building
Door Code 3344
SUNDAY, October 23rd to FRIDAY, October 28th
MONDAY, December 3rd to Saturday, December 10th

Above are my dates for October / December 2022

For those who already have an appointment –Thank You

Those who don’t please consider it or next time around

**Use the new & improved website as the# 1 referral tool

For those who have referred  thank you for trusting me

For the new patients–Welcome!

I spend quality time with each patient;getting to know what is going on with them;

and doing all within reason to bring them forward to a healthier stature

Mild Silver Protein is Back and Available

Mild Silver Protein is Back and Available

                     Mild Silver Protein

The untimely death of Bill McFarland earlier this year left a gaping hole in the availability of the Silver 500. He was the sole owner of MSP Research which distributed the silver under his name but was not the origin nor the manufacturer of it.

The origin of the silver including where it was made; how and who it was distributed to; and from where it was shipped had been under wraps due to the possibility of government interference in anything that was not pharmaceutical in nature—let alone guaranteed to work

As it turns out, I was given access to the identical Silver 500 ppm Bill McFarland used along with additional strengths such as 5000 ppm and 10000 ppm for conditions that are chronic and long term. The 5000 ppm may be the new “go to” strength.

When it concerns my patients and as with all products originating with me, I prefer to speak with you directly; determine what is appropriate based on my experience; and place the order myself.  Additionally, I am a big believer in having a few bottles of silver on hand for “just in case” purposes.

Prices vary according to Strength

Silver is 100% Non-Toxic / Self Sterilizing



  • Cool Mist Humidifier (Walmart $49.99)
  • 1 Gallon Distilled Water–MUST be Distilled
  • 1 Quart bottle of Hydrogen Peroxide
  • 18-20 drops Oregano Oil
  • 15 cc/1 Measuring Tablespoon of Silver 500 ppm

Stir and blend well,. Use when feeling stuffy/congested, manifesting cold virus symptoms, flu, coughing, or anything respiratory including nCOVID-19 symptomatology.


  • Fill cup 1/2 to 3/4 full of Silver 500 ppm
  • Add 2 drops of DMSO if available
  • OR if by chance you happen to have Albuterol-(call to discuss how to use for this recipe)
  • Do a breathing session

The best nebulizer for your money is made by InnoSpire Elegance Compressor Nebulizer System  Purchase from Just Nebulizers  


Ask about purchasing extra disposable and non-disposable mouthpieces. 

Have extra on hand, not just because of nCOVID-19, but also because of the many other respiratory issues.

NOTE: NEBULIZING silver is THE MOST EFFICACIOUS TREATMENT when it comes to preventing the progression of, and in the treatment of, pneumonia and respiratory distress.

The Canary in the Coal Mine -or- How to Improve Kidney Function

The Canary in the Coal Mine -or- How to Improve Kidney Function

By Dr. Douglas Lobay, BSc., ND

Improving kidney function can be difficult and exasperating. As a practicing naturopathic physician, I am always interested in improving the health of my patients. True to the tenets of do no harm, treat the whole patient, and stress preventative medicine, I am looking for ways to improve kidney function in my patients. I have become particularly interested in variations of creatinine and glomerular filtration rate and declining levels of function at the stage 2 and stage 3 kidney disease. I have been fortunate to practice chelation therapy, and I have monitored kidney function in hundreds of patients. Over the years I have analyzed many blood chemistry panels and performed countless urinalyses in the office. I have evaluated kidney function based on serum creatinine levels and estimated glomerular filtration rate (eGFR). Through continuity of care I have compared lab values over time for many patients and tried to figure out what improves kidney function and what causes its decline. Like the canary in the coal that is sent down into mine shafts to sniff out toxins before the miners are sent in, finding ways to improve kidney function before irreversible kidney damage occurs can be indemnifying.

I found Dr. Jenna C. Henderson’s article in the June 2019 Townsend Letter, titled “Is This Actually Kidney Disease, and What Can Be Done About It?” Here is a summary of important points that I learned from this article. Glomerular filtration rate (GFR) is an estimate of kidney filtration. It is associated with serum creatinine levels and also age, gender, and race. It is underestimated in the very young and the very old. If eGFR is decreased, then the first question to be asked is how well hydrated is the patient? Then the next question to be asked is does the patient have high blood pressure or hypertension? Serum creatinine levels can be increased with an increase in muscle mass. Blood urea nitrogen of BUN is a nitrogenous waste product that correlates with dietary protein intake. Renal anemia is due to decreased erythropoietin levels. Potassium levels may be increased with a decreased eGFR. The normal level of urinary pH is between 5.5 and 6.5. There are approximately one million nephrons in the kidneys of the adult human. You lose about 1% of nephrons per year with aging. Blood tests can detect serum creatinine levels, which can be used to estimate glomerular filtration rate. An increase in serum creatinine is associated with a decrease in GFR. Hyperfiltration of the glomerulus is associated with chronic kidney disease. Recovery from a decreased GFR and chronic kidney disease can be limited due to fibrosis of kidney tissue. Active cell division can promote kidney recovery.

Dr. Henderson further describes kidney supplements that may be beneficial for urinary tract infections or UTI’s, kidney stones, and chronic kidney disease. She likes animal studies that use the 5/6 nephrectomy model, in which 5/6 of the kidney is removed. The effect of a treatment can be measured in the remaining 1/6 of the functioning kidney. Dr. Henderson describes the kidney friendly diet. She also suggests the use of dietary supplements that have been shown in some studies to improve kidney function. Some of the supplements mentioned in this article include Epimedium sagittatum, resveratrol, hibiscus, Salvia miltiorrhiza, ubiquinol, citrus bioflavonoids, curcumin (especially tetrahydrocurcumin), and medicinal mushrooms like cordyceps. These supplements can help to ameliorate oxidative stress, inflammation, proteinuria and progressive renal damage.

I am not a kidney specialist, but I have an extraordinary appreciation of the nurses and doctors who work in kidney dialysis units. However, I am interested in how to improve kidney function in early kidney decline such as with stage 2 and 3 kidney disease on an outpatient basis. Stage 2 kidney disease is defined as an eGFR between 60 to 89 milliliters per minute, and stage 3 kidney disease is defined as an eGFR between 30 and 59 milliliters per minute. I decided to do a literature search on PubMed about natural ways to improve kidney function. I discovered that a good place to begin in understanding kidney disease is a thorough review of basic renal physiology.

Kidney Physiology Review

The kidneys are a pair of bean-shaped organs that are normally found in the right and left of retroperitoneal space in the abdomen. The average size of the right kidney is 10.9 centimeters in length and 11.2 centimeters for the left kidney. The average female kidney weighs 115 to 155 grams while the average male kidney weights 125 to 175 grams. Each kidney is supplied by one renal artery and one renal vein. The main function of the kidney is to filter the blood, maintain serum osmolarity, maintain electrolyte and acid/based balance, and remove toxins from the blood. The four mechanisms by which the kidneys work are filtration, re-absorption, secretion and excretion.

The functional unit of the kidney is the nephron. Each kidney has about one million nephrons. Hydrogen, ammonia, potassium, and uric acid are excreted from the nephrons; and water, sodium, bicarbonate, glucose, and amino acids are reabsorbed. Vitamin D or calcitriol is produced in the kidney, and hormones erythropoietin and rennin are also produced there. Each kidney is divided into an outer cortex and an inner medulla. Nephrons, primarily the glomeruli are located in the renal cortex. The afferent arteriole supplying prerenal blood enters the bulbous capsule of the nephron called Bowman’s capsule. The conglomeration of afferent arterioles inside Bowman’s capsule is called the glomerulus. The increased hydrostatic pressure of the afferent arteriole forces blood at high pressure into the glomerulus. Due to the hydrostatic pressure and membrane permeability, low weight molecular molecules are filtered across the capsular membrane. The blood is also filtered across endothelial cells of the arteriole, the basement membrane, podocytes, and around mesangial cells. An amazing one-fifth of the entire human blood supply is filtered each minute. An incredible amount of 180 liters of filtrate is produced each day through the renal glomerulus. The efferent arteriole leaves Bowman’s capsule with the remaining filtered blood. Meanwhile, the filtrate produced across the glomerulus drains down across a series of tubules, namely the descending proximal tubules, loop of Henle, the ascending tubules and the distal convoluted tubules and collecting ducts. The early proximal tubule reabsorbs glucose, amino acids, sodium, chloride, phosphate and water. The loop of Henle is a u-shaped bend of tubules that includes the thin descending loop and a thicker ascending loop. The thin descending loop of Henle reabsorbs mainly water that leads to concentration of urine produced. The thick ascending loop of Henle reabsorbs electrolytes including mainly sodium, 3 potassium chloride, magnesium, and calcium. The distal convoluted tubules reabsorb mainly sodium and chloride and the collecting ducts also reabsorb sodium and water. The urine filtrate produced leaves the collecting tubules through the renal medulla and ultimately through the calyces to the ureters and bladder.

Creatinine is a breakdown product of creatine phosphate found in muscles and is usually produced at a fairly constant rate and is somewhat dependent on muscle mass. About 1% to 2% of muscle creatine is converted to creatinine on a daily basis. It is a nitrogenous compound removed by the body primarily through glomerular filtration and a small amount through the proximal tubules. Men, through increased muscle mass, have higher creatinine levels than females. Serum creatinine levels are 0.5 to 1.0 milligrams/deciliter or 45 to 90 micromoles/liter in females and 0.7 to 1.2 milligrams/deciliter or 60 to 110 micromoles/liter in males. Lab values of creatinine also vary slightly from lab to lab. Creatinine has also demonstrated antibacterial and immunosuppressive properties. Creatinine levels can also be increased with high protein dietary consumption. Creatine clearance is associated with glomerular filtration rate. False elevations in serum creatinine can occur. Factors that can increase serum creatinine include race, age, body size, gender, menstruation, diurnal variation, intense exercise, and increased protein intake. Approximately 15% of creatinine is secreted by the renal tubules. Certain drugs can increase serum creatinine. These drugs include Trimethoprim, cimetidine, and H2 blockers. Rhadmyolysis or acute muscle damage can also increase creatinine levels.

Urea is a nitrogenous by byproduct of muscle metabolism produced in the urea cycle by conversion of nitrogen from amino acids to ammonia compounds. BUN production is somewhat more variable than creatinine and less reliable as an indicator of kidney filtration. Urea is increased from a diet rich in protein. The BUN to creatinine ratio provides some interesting information about the possible location of kidney dysfunction. A BUN to creatinine ratio greater than 20:1 indicates a prerenal cause. A BUN to creatinine ration between 10-20:1 indicates a post-renal cause. And a BUN to creatinine ratio of less than 10:1 indicates an intra-renal cause.

Glomerular filtration is an estimate of kidney function and degree of kidney disease. Glomerular filtration or GFR represents the flow of plasma into Bowman’s space in a specific period of time. Kidneys receive 20 to 25% of blood volume cardiac output equaling 1.0 to 1.2 liters per minute. Glomerular filtration rate is correlated with serum creatinine levels, age, body size, race, and gender. Stage 1 kidney disease has a GFR greater than 90 ml/minute and indicates normal kidney function. Stage 2 kidney disease has a GFR between 60 to 89 ml/minute indicates mild loss of kidney function. Stage 3a kidney disease has a GFR between 45 to 59 ml/min and indicates a mild to moderate loss of kidney function. Stage 3b kidney disease has a GFR between 30 to 44 ml/minute and indicates moderate to severe loss of kidney function. Stage 4 kidney disease has a GFR between 15 to 29 ml/minute and indicates severe loss of kidney function. Stage 5 kidney disease has a GFR less than 15 ml/minute and indicates severe kidney failure. Many labs use an arbitrary level of GFR less than 60 ml/minute to indicate kidney disease. Also the degree of protein or albumin in the urine has been correlated with the degree of loss of kidney function.

Hormones affect reabsorption of different molecules in the kidneys. Anti-diuretic hormone or ADH, produced in the posterior pituitary gland in the brain, is also known as vasopressin and stimulates water reabsorption through the kidney tubules. Vasopressin goes to the distal and collecting tubules and promotes the reabsorption of water back into circulation. A low blood pressure can stimulate the release of vasopressin into systemic circulation. Copeptin derived from the c-terminal is a surrogate marker of vasopressin and is more stable and measurable than ADH. Aldosterone produced in the adrenal glands stimulates sodium reabsorption through the kidney tubules. Parathyroid hormone, produced in the parathyroid glands, alters phosphate and calcium reabsorption.

Chronic kidney disease or CKD affects about 10 to 15% of the adult population of Western countries. It is a major risk factor for cardiovascular disease and death. CKD has been associated with hypertension, diabetes, and atherosclerosis. Structural and functional abnormalities of renal tissue show up in just three months after declining kidney function. There can be a genetic component to kidney pathology and kidney disease. Kidney disease can be further divided depending on the deposition of immune globulins. Proliferative kidney disease is an immune-dependent pathological process that results in inflammation and the deposition of immune complexes in the glomerulus and tubules. Non-proliferative kidney disease is not caused by immune globulin deposition and is not immune dependent. The podocyte of the glomerulus plays a central role in kidney disease pathology. Immune complexes can be deposited in mesangial cells of the glomerulus.

Chronic kidney disease is associated with renal fibrogenesis causing glomerular sclerosis and tubular interstitial fibrosis. Fibrogenesis induces inflammatory changes in the glomerulus and tubules. Inflammatory filtrate, profibrotic cytokines, and fibroblast proliferation occurs. Tubulo-interstitial inflammatory infiltrates include proteinuria, immune deposits, chemokines, calcium phosphate, metabolic acids, including uric acids and oxidized lipids. The net effect is that these deposits further caused renal deterioration.

Non-steroidal anti-inflammatory drugs decrease kidney prostaglandins. A decrease in prostaglandin synthesis, which regulates vasodilation at the glomerular level, causes a decrease in renal blood flow. This disrupts compensatory vasodilation causing renal vasculature constriction. NSAIDS also cause increased pressure in the renal interstitium for up to one week after starting these drugs. About 15% of acute kidney injury is due to NSAID consumption.

Diet and Kidneys

There is a direct association between water intake and kidney function. Water intake is related to chronic kidney disease, polycystic kidney disease, and nephrolithiasis or kidney stones. Benefits have been noticed when urine output has been measured to be between 3 to 4 liters per day. Increased water intake blocks the renal vasopressin V2 receptor. A comprehensive literature review studied the effects of hydration on kidney function. Increased water intake decreases vasopressin levels. Hydration can improve kidney function in all forms of chronic kidney disease, recurrent renal calculi, and slow renal cyst formation.2 A beneficial effect on kidney function in patients who are at risk for development of chronic kidney disease was noted. Although increased hydration and reduced vasopressin secretion appear to slow progression in patients with chronic kidney disease, over-hydration can be detrimental in patients with end-stage kidney disease. Beneficial effects have been noted in patients with earlier chronic kidney disease, diabetic nephropathy, and nephrolitiasis.

A randomized controlled trial was conducted for one-year duration to study the effect of increased water intake on kidney function. Increased water intake was associated with a decreased concentration of calcium oxalate, calcium phosphate, and uric acid. Increased urine output between 2 to 2.5 liters per days was a consequence of increased water intake. Increased water intake was associated with a decreased growth rate of polycystic kidneys. Six hundred and thirty-one participants with stage 3 chronic kidney disease and a GFR between 30 to 60 ml/minute were selected for this study. The participants were coached to increase water intake by 1.0 to 1.5 liters per day (4 to 6 cups of water in addition to other beverages usually consumed). Plasma copeptin levels were directly correlated with anti-diuretic hormone or vasopressin levels. Vasopressin was the first hormone released during the early stages of dehydration. The increased level of vasopressin was correlated with early kidney disease. Other surrogate markers, including estimated glomerular filtration rate, copeptin, and microalbuminuria, were measured. Increased water intake was associated with improved kidney function markers.

Fluid self-management recommendations were made for prevention of kidney stones based on epidemiological evidence. A urine fluid output of 2.5 liters per day or more was recommended. Potassium-rich foods protected against kidney stone formation. Increasing fluid intake of 500 ml of water per day decreased kidney stone formation. Tea and alcohol consumption also decreased kidney stone formation. Fruit juice, soda and pop did not decrease kidney stone formation and were suggested to increase rates. Coffee and milk showed inconsistent results and could not be recommended as a fluid to decrease kidney stone formation. The combined interaction of environmental exposure, dietary habits, and genetic factors cause kidney stones. Water intake is recommended to be three liters or greater per day to help prevent kidney stones. It is not the quality of water, rather the quantity of water consumed that matters in most cases. The correlation between water hardness, which increased mineral concentration, and the development of kidney stones remains controversial.

Low-protein and plant-based diets are beneficial in chronic kidney disease. Low protein diets can reduce protein intake, phosphorus and sodium. Plant-based diets are low in saturated fats, high in fiber, and high in unsaturated fats and potassium. These diets are low in creatine.

High salt intake can have detrimental effects on kidneys by affecting glomerular function. Increased glomerular hyper-filtration, increased filtrate formation, and increased glomerular pressure are a consequence of high-salt consumption. Increased sodium intake blunts the anti-proteinuria effects of various drugs, including ACE inhibitors. Increased sodium further causes increased blood pressure, increased renal hypertrophy, renal fibrosis, decreased glomerular basement membrane, and decreased anionic membrane function. Restricting sodium intake is an important preventable measure in patients with chronic kidney disease.8

A modest coffee intake of one to two cups per day decreased the risk of developing chronic kidney disease. The odds ratio of developing kidney disease was 0.76 with drinking one cup of coffee per day. The odds ratio of developing kidney disease was 0.80 by drinking two cups of coffee per day.9Decreased plasma creatinine levels were observed in mice that consumed decaf coffee for two weeks. High-dose decaf coffee increased nucleotide activation in the kidney cortex, which increased kidney function. Meta-analysis of four observational studies with 14,098 people showed no significant association between coffee intake and development of chronic kidney disease. Interestingly however, the odds ratio was 0.81 in females and 1.10 in males.

Green tea consumption was associated with a decreased risk of developing kidney stones in a large prospective study of elderly Chinese people. The odds ratio was 0.78 in males and 0.80 in females.1 Green tea polyphenols protected the kidneys against oxidative stress damage in early renal damage.

Apple cider vinegar induces a protective effect on oxidative damage to the kidney in ovariectomized mice fed a high-cholesterol diet.

Dietary treatment of urinary risk factors for renal stone formation was assessed. A general conservative therapy was recommended. Decreased fluid intake was associated with decreased urine output less than 2 liters per day. Dietary calcium supplementation was not recommended. Dietary calcium less than 1 gram per day, low protein diet, and low sodium diet were recommended. Mild dietary salt intake was associated with decreased calcium excretion. Calcium supplementation was recommended between 800-1200 mg per day. A low normal protein diet was associated with decreased calcium loss. Alkaline citrate minerals were recommended. Increased fruit and vegetables excluding high oxalate food were suggested. Increased consumption of citrate foods and melons increased urinary citrate levels.

Vitamins, Minerals, and Kidneys

Oxidative stress is a disruption of the balance between the production of reactive oxygen species or ROS and antioxidant defense mechanisms. Oxidative stress was identified in early kidney disease. Suggested potential useful antioxidants have been identified in food and supplements. Some proposed antioxidant supplements include vitamin B, C, D and E, coenzyme Q10, L-carnitine, alpha lipoic acid, curcumin, green tea, flavonoids, polyphenols, omega-3 polyunsaturated fatty acids, statins, trace elements and n-acetyl cysteine.16

In a double-blind randomized controlled trial, 60 patients with diabetic nephropathy were supplemented with 1200 IU of vitamin E or placebo for 12 weeks. Vitamin E supplementation was observed to increase serum levels. Decreased protein to creatinine ratio, decreased tumor necrosis factor or TNF, decreased matrix metalloproteins, and decreased malondialdehyde levels were noted. No effects on other biomarkers of kidney disease were observed. Decreased levels of inflammation, kidney injury, and oxidative were also observed.17 Vitamin E supplementation of 300 milligrams per day for 12 weeks with 20 diabetic and 20 non-diabetic patients with end stage kidney disease undergoing hemodialysis. Vitamin E supplementation improved HDL levels and vascular endothelial function.18

High homocysteine has been correlated with kidney disease. Low levels of vitamins B12, B9, and B6 have been associated with high homocysteine levels. One hundred and forty-eight 7 patients with chronic kidney disease from the Ukraine were measured for homocysteine levels; 58.7% of patients had high homocysteine levels. Homocysteine was believed to be a specific indicator of low vitamin status, especially for vitamin B9 or folic acid.19

Recurrent kidney stone formation in patients with normal renal function who have a defect in ascorbic acid to oxalate metabolites should restrict vitamin C supplementation to less than 100 mg per day. However, a large-scale prospective study showed that the group with the highest quintile of vitamin C intake, which was greater than 1500 mg/day, had the lowest risk of kidney stone formation.

Long-term intake of vitamin D resulted in an increased risk of hypercalcemia and hypercalciuria, which appeared to be not dose related. However, vitamin D supplementation did not increase the risk of kidney stone formation. It was recommended that large randomized controlled trials of vitamin D are needed to confirm these findings.

Magnesium is utilized for over 300 different biochemical reactions in the human body. Magnesium helps to decrease vascular calcium and decrease phosphate absorption.

Supplements and Kidneys

Coenzyme Q10 is a strong antioxidant and mitochondrial nutrient. Decreased Q10 levels have been observed in patients with chronic kidney disease. A meta-analysis of seven randomized controlled trials of patients with chronic kidney disease were selected from 721 potential papers from PubMed, Cochrane, and other medical databases were selected. Coenzyme Q10 supplementation was associated with decreased total cholesterol, decreased LDL cholesterol, decreased malondialdehyde, and decreased creatinine levels. Triglyceride levels as well as HDL cholesterol, glucose, insulin and CRP levels were not affected by Q10 supplementation.24 The effect of coenzyme Q10 supplementation was studied on 72 rats exposed to radiation-induced nephropathy. Q10 was administered at a dose of 10 mg/kg body weight. Signs of poor kidney function were observed after radiation exposure. Q10 supplementation decreased BUN and creatinine levels. Q10 seemed to attenuate glomerular and tubular function in irradiated kidneys. Administration of Q10 seemed to alleviate radiation induced kidney damage.25 The effect of benfotiamine, a derivative of vitamin B1 or thiamine and co-enzyme Q10 was studied a group of rats that had gentamycin-induced kidney damage. Gentamycin was severely toxic to rats. Benfotiamine supplementation decreased nephrotoxicity, decreased BUN and creatinine levels and decreased malondialdehyde levels. Q10 supplementation decreased necrotic tubular tissue levels and hyaline accumulation.

Resveratrol is a natural polyphenolic compound found in grapes and other compounds. Resveratrol is a robust antioxidant that scavenges reactive oxygen species or ROS. Resveratrol is also a SIRT-1 activator, which increases enzyme functions, decreases apoptosis, and increases mitochondrial biogenesis. Resveratrol has demonstrated to have some renal protective effects.

N-acetyl cysteine or NAC administration ameliorates renal function by decreasing oxidative damage as observed in Wistar rats with induced kidney damage following bilateral ureteral obstruction.

Acetyl-l-carnitine improved energy metabolism in the kidneys of hypoxic-ischemic brain-injured rats.

L-carnitine levels have been observed to be low in patients with advanced kidney disease. A systematic review of the evidence of carnitine and kidney disease was undertaken. Fourteen references were identified for review. A meta-analysis shows some useful pieces of information and the potential benefit of carnitine supplementation in patients with advanced kidney disease. The authors recommend carrying out better designed studies to show the potential benefits of this supplement.

Lipoic acid is a free radical scavenger of reactive oxygen species (ROS) and nitric oxide (NO). It’s mechanism of action is not completely understood but is believed to operate with different kinase pathways. Lipoic acid is known to affect multiple signaling pathways and has demonstrated renal protective effects in contrast-induced kidney injury.

Melatonin prevents drug-induced renal injury by decreasing inflammatory cytokine production and decreased aquaporin water channels in male rat’s kidneys with bilateral ureteral obstruction.32

Herbs and Kidneys

Berberine showed antioxidant, anti-apoptotic, and anti-inflammatory activity in diabetic rats exposed to nephrotoxic streptozotocin. Decreased levels of BUN and creatinine were observed with berberine doses of 100 and 150 milligrams per kilogram. Improved histopathological kidney function was also observed with declining levels of BUN and creatinine. Increased free fatty acids and disturbed mitochondrial function play a role in diabetic kidney disease. Diabetic mice were fed berberine for eight weeks. Berberine reversed glucose and lipid metabolic podocyte damage, basement membrane thickening, mesangial expansion and glomerular sclerosis. Decreased podocyte apoptosis was also observed.34 Berberine demonstrated a protective effect on kidney and liver function in rats exposed to iron overload for two weeks. Increased iron consumption increased oxidative damage causing histopathological changes to these organs. Elevated levels of oxidative damage also increased malondialdehyde levels, which is a marker of oxidative damage. Berberine protected the liver and kidney against ferrous sulphate-induced toxicity, reduced lipid peroxides, and also chelated iron.35 Chronic diabetic kidney disease causes microvascular damage to the renal glomerulus and causes interstitial fibrosis to the renal tubules. Berberine showed a significant positive effect by decreasing total blood sugar levels, improving renal hemodynamics, improving lipid profiles, and was shown to attenuate local and systemic inflammation.

Berberine protects renal tubular cells from ischemia and reperfusion injury in rats. Berberine protects renal tubular epithelial cells from hypoxia and reperfusion mitochondrial dysfunction by regulating sirtuin-2 and c53 pathways.37 Berberine also protects renal tubular epithelial cells from hypoxia and apoptosis through activation of HIF-1alpha in the signaling pathway suggesting that berberine could be a potential drug in diabetic ketoacidosis.

Hydrangea or Hydrangea paniculata extract was given to mice with cisplatin-induced kidney damage. Improved renal function was observed with decreased creatinine and BUN levels. The coumarin glycosides of this plant were believed to attenuate oxidative stress, decrease tubular injury and decrease apoptosis. Decreased inflammatory cytokines and regulation of protease enzymes, including caspase, an enzyme involved in inflammation and programmed cell death, were noted. Hydrangea extracts ameliorated kidney damage by antioxidant activity and suppressing harmful renal infiltrates and tubular apoptosis. Decreased renal infiltrates down regulated signaling pathways, which further decreased macrophage and neutrophil activation.39

Hydrangea extracts containing coumarin were given to mice with acute septic kidney injury. Bioactive coumarin compounds isolated from Hydrangea paniculata were identified to be umbelliferone and esculetin. Hydrangea demonstrated specific antioxidant and anti-inflammatory activity on kidney tissue. Decreased infiltrate of macrophages and neutrophils in renal infiltrate was noted. Decreased BUN levels and decreased tubular interstitial injury were noted. Improved kidney function and increased animal survival were observed.40 Water extracts of Hydrangea paniculata were rich in coumarin glycosides. A water extract of this plant was given to diabetic rats that were exposed to strepozotocin kidney damage. Hydrangea extracts were given once per day for three months. A decrease in BUN and creatinine was observed. The hydrangea extract upregulated some enzyme activity and decreased other signaling pathways.

Silybin from milk thistle (Silybum marianum) protected against cisplatin-induced acute kidney injury. Silybin increased sirtuin-3 or Sirt-3 expression in mice, which was shown to protect against cisplatin-induced kidney damage by decreasing tubular apoptosis and increasing mitochondrial function. A standardized mixture of silymarin flavonolignans from milk thistle has demonstrated antioxidant activity for reactive oxidant species (ROS). Contrast-induced nephropathy caused tubular injury in mice kidneys. Silymarin preserved renal function by improving glomerular and tubular function in a dose dependent manner in mice exposed to contrast injury.

Apigenin is a flavonoid isolated from celery (Apium graveolens). Apigenin is an anti-hypertensive flavone that is abundant in celery. Apigenin acts as an agonist of transmut receptor-potential vanilloid-4 or TRPV4 in rats. Apigenin has demonstrated promising anti-hypertensive activity in treating hypertensive-induced renal damage in those who consume a high sodium diet.44 A hydroalcoholic extract of celery was used to treat uncomplicated urinary tract infection in mice caused by uropathogenic E.coli. A direct anti-adhesive effect of celery on the urinary epithelium was observed in a dose dependent manner within four to seven days of treatment. The extract significantly reduced the bacterial load in uncomplicated UTIs in this group of mice.45 In another study, rats were exposed to a chlorinated propanediol chemical that induced kidney damage. Apigenin decreased serum creatinine and reduced kidney damage by modulating mitochondria dependent caspase activity.46

An herbal decoction of the Chinese medicine Huangqi-Danshen was fed to a group of rats for four weeks. Improved levels of BUN and creatinine were observed after treatment. Decreased tubular atrophy and decreased interstitial fibrosis was also observed. Also improved mitochondrial function was also observed in kidney tissue of these animals.47 A water extract of 10

Salvia miltiorrhiza called Danshen was studied against renal injury in rats exposed to cadmium. The salvia extract displayed antioxidant activity, decreased swelling of renal tubule epithelial cells, and improved overall renal function.48 Salvia miltiorrhiza extract was given to diabetic rats and was shown to decrease renal injury and damage and improve glycolipid metabolism. Multi-modal effects were observed, including phospholipid, arachidonic acid, wnt/B-catenin and TGY-B signaling inhibition.49 Salvianolic acid A (SAA) is a phenolic carboxylic acid derivative of Salvia miltiorrhiza. SAA helped to decrease inflammation and decrease renal tubular fibrosis in six rats that were exposed to doxorubicin-induced nephrotoxicity. SAA was believed to be a signaling molecule that decreases activation of natural factor beta-kappa (NF-KB) and thereby decreases inflammatory pathways.50

An aqueous extract of parsley (Petroselinum sativum) was studied on a group of rats. The parsley extract showed diuretic effects including decreased sodium reabsorption, decreased potassium secretion, increased potassium concentration in the intercellular space, and decreased potassium efflux across cellular tight junctions. An inhibition of sodium/potassium pump activity was noted leading to a reduction of sodium reabsorption and potassium loss.51 The effects of an aqueous extract of parsley on ethylene glycol-induced kidney calculi in rats were studied. A decrease in calcium oxalate stone formation and deposition were noted.52.

The effect of Diyarbakir watermelon juice on lipid peroxidation was studied in rats after exposure to carbon tetrachloride. The watermelon juice showed antioxidant activity and inhibition of lipid peroxide formation.53 However, excessive watermelon consumption was associated with high potassium levels or hyperkalemia and increased symptom burden of end stage renal disease.54 The anti-urolithiatic and diuretic effects of watermelon (Citrullus lanatus) was studied in a group of male rats. Kidney protective effects and a suppression of calcium oxylate precipitation were noted. Increased creatinine clearance, decreased urea, decreased sodium, and urinary output were also observed. Watermelon pulp displayed significant anti-urolithic and diuretic activity.55

Thirty-four neutered cats were put on a nutraceutical diet containing Lespedeza, Vaccinium and Taraxacum herbs for 90 days. There was a significant decrease in BUN, creatinine in lab samples and a decrease in total protein lost from their kidneys.56 Researchers compared the effects of taraxasterol aqueous extract from dandelion (Taraxacum officinale) aerial parts and potassium citrate on oxylate crystallization in vitro. Taraxasterol and potassium citrate extract decreased calcium oxylate crystallization more than taraxasterol alone. It was hypothesized that there was a synergistic effect between Taraxacum extracts and potassium citrate.57

The effect of green asparagus (Asparagus officinalis) extract was studied in hypertensive rats. A constituent of asparagus, 2’-hydroxynicotianamine, was believed to act as an angiotension converting enzyme or ACE inhibitor. The asparagus extract decreased blood pressure levels and improved renal function.58 The effect of asparagus extract was studied in bisphenol-causing liver and kidney damage in Wistar rats. Coadministration of asparagus extract and bisphenol exposure increased total antioxidant capability and decreased malondialdehyde levels, which is indicator of oxidative stress. Decreased levels of oxidative damage to liver and kidneys with asparagus consumption were noted.59 11

Black seed (Nigella sativa) has been used in the prevention of kidney stones. In a placebo controlled double-blind study 60 patients consumed 500 mg of black seed or placebo twice per day for 12 weeks. Those who consumed black seed were 44.4% more likely to excrete kidney stones compared to 15.3% who took placebo. The black seed-treated group showed a decrease in the size of kidney stones in 51.8% of people while stone size was unchanged in 57.6% of those who took a placebo.60 The effect of black seed oil was studied in cisplatin-induced nephropathy in rats. The group that consumed black seed oil showed a marked decrease in histopathological changes to kidney tissue. Black seed displayed antioxidant activity and was shown to modulate enzyme activity in the kidney.61

The long-term survival rate was studied in retrospective analysis of Taiwan patients with chronic kidney disease who took a Chinese herbal medicine containing Rheum officinale. The Chinese herbal medicine improved long-term survival in patients with CKD suggesting that this medicine is an effective adjuvant in individuals with chronic kidney disease.62

An aqueous extract of hibiscus (Hibiscus sabdariffa) was given to rats for 28 days with adenine-induced kidney disease. The hibiscus extract demonstrated a similar effect to the angiotensin converting enzyme or ACE inhibitor drug lisinopril. The hibiscus extract was further demonstrated to decrease the progression of chronic kidney disease.63

Goldenrod (Solidago virgaurea) has been used for centuries for the treatment of urinary tract disease. Goldenrod has displayed anti-inflammatory, anti-urothiac, diuretic, anti-spasmodic, and analgesic effects. Goldenrod has been used for urinary infections and inflammation, to prevent kidney stone formation, and to remove urinary gravel. It appears to be safe and non-toxic and no adverse side effects were noted.64

Curcumin was reported to have kidney protective effects.65 Four hundred and fourteen patients with chronic kidney disease with a glomerular filtration rate between 15 to 60 ml/min were included in this randomized controlled trial. Patients consumed 90 milligrams of micro-particle curcumin once daily or placebo for six months.

Butein from Chinese lacquer tree (Rhus verniciflua) has been shown to improve kidney function in cisplatin-induced acute renal failure in rats. Butein upregulates aquaporin 2 channels in the kidney cortex and medulla.66

Other herbal medicines have also been suggested to improve kidney function, including beets, chamomile, Chanca Piedra, cinnamon, ginger, gravel root, horsetail, marshmallow, nettles, and uva ursi to name a few.

Toxins and Kidneys

Certain dietary supplements and herbs have been identified to cause potential kidney toxicity and renal damage. Implicated herbs include Chinese yew (Taxus celebica), Callilepsis laureola, morning cypress (Cupressus tunebris), Saint John’s wort (Hypericum perforatum), Thunder God vine (Tripteryglum wildfordi), tribulus (Tribulus terrestris), and wormwood (Artemnesia alba). Herbs no longer sold in the US include chocolate vine (mu tong or Caulis aristolochiae), Guang fang ji (Artistolochia fungchi), and Ma huang (Ephedra sinensis). Other dietary supplements include sheep bile, chlorella, chromium, creatine, fish gall bladder, glucosamine, hydrazine, NO-xplode, Spanish fly, and excessive consumption of vitamins A, C, D and germanium. In descending order of toxicity, the top two offenders were aristolochic acid in the herb Guang fang ji and chocolate vine. Kidney toxic foods include ajenkol bean, gall bladder from carp fish, puffer fish, star fruit, and uncooked yam powder or juice.

Aristolochic acid from Aristolochia species of herbs causes nephropathy. In the 1990s aritstolochia was used in various weight-loss products. Consumption of products containing aristolochic acid causes kidney injury and renal damage. Increased serum creatinine, significant anemia, and histopathological changes in renal interstitial infiltrates causing severe fibrosis occurred.68

Chronic kidney disease is associated with an increased level of uremic toxins. Certain toxins may be modified by the gut microbiota. Potential toxins identified include phosphates, l-carnitine, choline, phosphatidylcholine, tryptophan and tyrosine, tri-methylamine-n-oxide, indoxy sulphate, p-cresyl sulphate, and indole-3 acetic acid.69


It is clear that diet and nutrition plays an important role in kidney function. Water intake can be measured and increased in individuals with stage 2 and 3 chronic kidney disease. There are exceptions to increased water intake especially with people with lung and peripheral edema. Fluid restrictions are further necessary in individuals with advanced kidney disease, including stage 4 and 5 levels. Salt and sodium should be restricted in usually all levels of kidney disease, especially with high dietary intake. Protein intake can be measured and modulated in individuals with high dietary intake. Electrolyte levels, including potassium and phosphorus, can be monitored and measured along with creatinine and GFR in basic lab tests. Recommendations can be made after reviewing lab values. Eating a diet rich in fresh fruits, vegetables, whole grains and cereals and high-quality protein is strongly suggested. Limiting processed foods, highly refined sugar foods, excessive protein and alcohol is also suggested. Eating better can perhaps help kidney function and improve overall health.

A review of the scientific literature suggests that vitamins and minerals, supplements and herbal medicines may help to maintain and, in some cases, improve kidney health. Many of the studies are done on animal models that include rats and mice. While interesting it is important to note that these are not humans. There are some human studies that are interesting and show benefit, but it clear that most of the conclusions are preliminary at best. Further randomized double blind placebo-controlled trials are suggested with larger patient populations and limiting extraneous factors. This is not to say that certain nutrients and supplements are not beneficial and cannot actually improve kidney function. There are many vitamins, minerals, supplements and herbal medicines as discussed in this article that have been shown to prevent kidney decline and may improve renal function. How they combine with other nutrients is not known but probably not harmful in recommended doses. They are certainly worth a try with individuals with declining kidney function, especially earlier in stage 2 and 3 levels of kidney disease. Discussing a treatment plan with a licensed naturopathic physician or other qualified health professional is recommended. Of course, it is caveat emptor or patient trial and error. Like the canary in the coal mine, I think kidney function decline with early kidney disease can be averted with dietary changes and nutritional supplementation.

Dr. Douglas Lobay, BSc, ND

Douglas G. Lobay is a practicing naturopathic physician in Kelowna, British Columbia. Dr. Lobay graduated with a bachelor of science degree from the University of British Columbia in 1987. He then attended Bastyr College of Health Sciences in Seattle, Washington, and graduated with a doctorate of naturopathic medicine in 1991.

While attending Bastyr College, he began researching the scientific information on the use of food, nutrition, and natural healing. Dr. Lobay enjoys research, writing, and teaching others about good health and good nutrition.

He is the author of four books and numerous articles in magazines. He also enjoys hockey, skiing, hiking, tennis, and playing guitar.


1. Wang CJ et al. The medicinal use of water in renal disease. Kidney Int. 2013 Jul:84(1): 45-53.

2. Clark WF et al. Hydration and Chronic Kidney Disease Progression; A Critical Review of the Evidence. Am J Nephrol. 2016: 43(4):281-92.

3. Clark WF et al. Hydration and Chronic Kidney Disease Progression: A Critical Review of the evidence. Am J Nephrol. June 2016;43:281-292. 14

4. Clark WF et al. The Chronic Kidney Disease Water Intake Trial: Protocol of a Randomized Controlled Trial. Can J Kidney Health Dis. 2017;4: 2054358117725106.

5. Chang Xu et al. Self-Fluid Management in Prevention of Kidney Stones: A PRISMA-Compliant Systematic Review and Dose-Response Meta-Analysis of Observational Studies. Medicine (Baltimore). 2015 Jul;94 (27): e1042.

6. Mitra P et al. Does the quality of drinking water matter in kidney stone disease. A study in West Bengal, India. Investig Clin Urol. 2018 May(3):158-165.

7. Post A et al. Creatine is a Conditionally Essential Nutrient in Chronic Kidney Disease: A Hypothesis and Narrative Literature Review. Nutrients. 2019 May 10:11(5).

8. Boero R et al. Salt intake and kidney disease. 2002 May-Jun:15(3):225-9.

9. Jhee, JH et al. Effects of Coffee Intake on Incident Chronic Kidney Disease: Community-Based Prospective Cohort Study. Am J Med. 2018. Vol 131 (12):1482-1490. December 2018.

10. Rybakowska IM et al. Effect of decaffeinated coffee on function and nucleotide metabolism in kidney. Mol. Cell Biochem. 2018 Feb:439(1-2):11-18.

11. Wijampreecha K et al. Association of coffee consumption and chronic kidney disease: A meta-analysis. Int J Clin Pract. 2017 Jan:71(1).

12. Shu X et al. Green tea intake and risk of incident of kidney stones: Prospective cohort studies in middle-aged and elderly Chinese individuals. Int J Urol. 2019 Feb: 26(2): 241-246.

13. Yi W et al. Green Tea Polyphenols Ameliorate the Early Renal Damage Induced by a High-Fat Diet via Ketogenesis/SIRT3 Pathway. Oxid Med Cell Longev. 2017: 9032792.

14. Naziroglu M et al. Apple cider vinegar modulates serum lipid profile, erythrocyte, kidney, and liver membrane oxidative stress in ovariectomized mice fed high cholesterol. J Membr Biol. 2014 Aug: 247(8):667-73.

15. Prezioso D et al. Dietary treatment of urinary risk factors for renal stone formation. A review of CLU Working Group. Arch Ital Urol Androl. 2015 Jul 7;87(2):105-120.

16. Liakopoulos V et al. Antioxidant Supplementation in Renal Replacement Therapy Patients: Is There Evidence? Oxid Med Cell Longev. 2019 Jan 15;20199109473. Ecollection 2019.

17. Khatami PG et al. The effects of high-dose vitamin E supplementation on biomarkers of kidney injury, inflammation, and oxidative stress in patients with diabetic nephropathy: A randomized, double-blind, placebo-controlled trial. J Clin Lipidol. 2016 Jul-Aug:10(4): 922-929. 15

18. Mune M et al. Vitamin E supplementation improves high density lipoprotein and endothelial functions in end-stage kidney disease patients undergoing hemodialysis. Clin Nephrol. 2018 Sep:90(3):212-221.

19. Shevchuk SV et al. The relationship between homocysteine level and vitamins B12, B9 and B6 in patients with chronic kidney disease. Wiad Lek. 2019;72(4):532-538.

20. Gerster H. No contribution of ascorbic acid to renal calcium oxalate stones. Ann Nutr Metab. 1997;41(5):269-282.

21. Malihi Z et a. Hypercalcemia, hypercalciuria, and kidney stones in long-term studies of vitamin D supplementation: a systematic review and meta-analysis. Am J Clin Nutr. 2016 Oct; 104(4):1039-1051.

22. Massy ZA et al. Magnesium-based interventions for normal kidney function and chronic kidney disease. Magnes Res. 2016 Apr 1:29(4):126-140.

23. Xu Y et al. Efficacy of coenzyme Q10 in patients with chronic kidney disease: protocol for a systematic review. BMJ Open. 2019 May 14:9(5); e029053.

24. Bakhshayeshkaram M et al. The Effects of Coenzyme Q10 Supplementation on Metabolic Profiles of Patients with Chronic Kidney Disease: A Systematic Review and Meta-analysis of Randomized Controlled Trials. Curr Pharm Des. 2018;24 (31):3710-3723.

25. Ki Y et al. Effect of Coenzyme Q10 on Radiation Nephropathy in Rats. J Korean Med Sci. 2017 May;32(5):757-763.

26. Ustuner MA et al. Effects of benfotiamine and coenzyme Q10 on kidney damage induce gentamicin. Tissue Cell. 2017 Dec;49(6):691-696.

27. Kitada M and Kova D. Renal Protective Effects of Resveratrol. Oxid Med Cel Longev. 2013: 568093. Published online 2013 Nov 28.

28. Shimizu MH et al. N-acetylcysteine protects against renal injury following bilateral ureteral obstruction. Nephrol Dial Transplant. 2008 Oct 23(10):3067-74.

29. Wang AG et al. Effect of Acetyl-L-carnitine Used for Protection of Neonatal Hypoxic-Ischemic Brain Injury in Male and Female Rats. Neurochem Res. 2019 Apr 30.

30. Gholipur-Shahraki T et al. Effects of Carnitine on Nutritional Parameters in Patients with Chronic Kidney Disease: An Updated Systematic Review and Meta-Analysis. J Res Pharm Pract. 2018 Apr-Jun;7(2):57-68.

31. Zhang J and McCullough PA. Lipoic Acid in the Prevention of Acute Kidney Injury. Nephron. 2016: 134(3):133-140. 16

32. Z Li et al. Melatonin therapy protects against renal injury before and after release of bilateral ureteral obstruction in rats. Life Sci. 2019 Jul 15: 229:104-115.

33. Kumas M et al. Investigation of dose-dependent effects of berberine against renal ischemia/reperfusion injury in experimental diabetic rats. Nefrologia. 2019 Jul-Aug:39(4).

34. Qin X et al. Berberine Protects Glomerular Podocytes via Inhibiting Drp1-Mediated Mitochondrial Fission and Dysfunction. Theranostics. 2019 Feb 28:9(6): 1698-1713.

35. Gholampour F and Keikha S. Berberine protects the liver and kidney against functional disorders and histological damages induced by ferrous sulfate. Iran J Basic Med Sci. 2018 May: 21(5):476-482.

36. Ren YL et ak, Research progress of berberine in the treatment of diabetic kidney disease. Zhongguo Zhong Yao Za Zhi. 2017 Feb;42(3):438-442.

37. Lin Y et al. Berberine protects renal tubular cells against hypoxia/reoxygenation injury view the Sirt1/p53 pathway. J Nat Med. 2018 Jun;72(3):715-723.

38. Zhang X et al. Protective effect of berberine on high glucose and hypoxia-induced apoptosis via the modulation of HIF-1a in renbal tubular epithelial cells. Am J Transl Res. 2019 Feb 15;11(2):669-682.

39. Sen Z et al. Total Coumarins from Hydrangea paniculata against Cisplatin-Induced Acute Kidney Damage in Mice by Suppressing Renal Inflammation and Apoptosis. Evid Based Complement Alternat Med. 2017;2017:5350161.

40. Zhang S et al. Total Coumarins from Hydrangea paniculata Show Renal Protective Effects in Lipopolysaccharide –Induced Acute Kidney Injury via Anti-inflammatory and Antioxidant Activities. Front Pharmacol. 2017 Dec 14;8:872.

41. Sen Z et al. Coumarin glycosides from Hydrangea paniculata slow down the progression of diabetic nephropathy by targeting Nrf2 oxidation and smad 2/3-mediated profibrosis. Phytomedicine. 2019 Apr;57:385-395.

42. Li Y et al. Activation of Sirtuin 3 by Silibin Attentuates Mitochondrial Dysfunction in CIsplatin-induced Acute Kidney Injury. Front Pharmacol. 2017 Apr 5;8:178.

43. De Souza Santos V et al. Silymarin protects against radiocontrast induced nephropathy in mice. Life Sci. 2019 Jul 1;228:305-315.

44. Wei X et al. Activation of TRPV4 by dietary apigenin antagonizes renal fibrosis in deoxycorticosterone acetate (DOCA)-salt-induced hypertension. Clin Sci (Lond). 2017 Apr 1;131(7):567-581. 17

45. Sarchar S et al. Antiadhesive hyrdoalcoholic extract from Apium graveolens fruits prevents bladder and kidney infection against uropathogenic E. coli. Fitoterapia. 2018 Jun;127:237-244.

46. Zhong Y et al. Protective effects of apigenin against 3-MCPD-induce renal injury in rat. Chem Biol Interact. 2018 Dec 25;296:9-17.

47. Liu X et al. Huangqi-Danshen Decoction Ameliorates Adenine-Induced Chronic Kidney Disease by Modulating Mitochondrial Dynamics. Evid Based Complement Alternat Med. 2019 Jan 1: 2019.9574045.

48. He L et al. Effects of water extract of salvia miltiorrhiza against renal injury in rats exposed to cadmium. Zhonghua Yi Xue Za Zhi. 2017 Jan 3:97(1):57-61.

49. Xiang X et al. Salvia miltiorrhiza protects against diabetic nephropathy through metabolome regulation and wnt/B-catenin and TGY-B signaling inhibition. Pharmacol Res. 2019 Jan:139:26-40.

50. Zhang HF et al. Salvianolic acid A attenuates kidney injury and inflammation by inhibiting NF-kB and p38 MAPK signaling pathways in 5/6 nephrectomized rats. Acta Pharmacol Sin. 2018 Dec:39(12);1855-1864.

51. Kreydiyyeh SI and Usta J. Diuretic effect and mechanism of action of parlsey. J Ethnopharmacol. 2002 Mar;79(3):353-7.

52. Saeidi J et al. Therapeutic effects of aqueous extracts of Petroselinum sativum on ethylene glycol-induced kidney calculi in rats. Urol J. 2012 Winter;9 (1):361-6.

53. Atlas S et al. Protective effect of Diyarbakir watermelon juice on carbon tetrachloride-induced toxicity in rats. Food Chem Toxicol. 2011 Sep;49 (9):2433-8.

54. Chan KY et al. Excessive watermelon consumption causing hyperkalaemia and increased symptom burden of an end stage renal disease patient. Nephrology (Carlton). 2016 Aug;21(8):721.

55. Siddiqui WA et al. Evaluation of anti-urolithiatic and diuretic activities of watermelon (Citrullus lanatus) using in vivo and in vitro experiments. Biomed Pharmacother. 2018 Jan; 97-1212-1221.

56. Di Cerbo A et al. A nutraceutical diet based on Lespedeza spp., Vaccinium macrocarpon and Taraxacum officinale improves spontaneous feline kidney disease. Physiol Rep. 2008 Jun:6(12) e13737.

57. Yousefi-Ghale Salimi et al. Inhibitory effects of taraxasterol and aqueous extract of Taraxacum officinale on calcium oxalate crystallization: in vitro study. Ren Fail. 2018 Nov; 49(1):298-305. 18

58. Sanae M and Yasuo A. Green asparagus (Asparagus officinalis) prevented hypertension by an inhibitory effect on angiotension-converting enzyme activity in the kidney of spontaneously hypertensive rats. J Agric Food Chem. 2013 Jun 12;61(23):5520-5.

59. Poormoosavi SM et al. Protective effects of Asparagus officinalis extract against Bisphenol A-induced toxicity in Wistar rats. Toxicol Rep. 2018 Mar 9;5:427-433.

60. Ardakani Movaghati MR et al. Efficacy of black seed (Nigella sativa) on kidney stone dissolution. A randomized double-blind, placebo-controlled trial. Phytother Res. 2019 May;33(5):1404-1412.

61. Foroqui Z et al. Protective effect of Nigella sativa oil on cisplatin induced nephrotoxicity and oxidative damage in rate kidney. Biomed Pharmacother. 2017 Jan;85:7-15.

62. Huang KC et al. Chinese Herbal Medicine Improves the Long-Term Survival Rate of Patients With Chronic Kidney Disease in Taiwan: A Nationwide Retrospective Population-Based Cohort Study. Front Pharmacol. 2018 Oct 1:9:1117.

63. Ali BH et al. Effects of aqueous extract and anthocyanins of calyces of Hibiscus sabdariffa (Malvaceae) in rates with adeinin-induced chronic kidney disease. J Pharm Pharmacol. 2017 Sep;69(9):1219-1229.

64. Melzig MF. Goldenrod—a classical exponent of urological phytotherapy. Wein Med Wochenschr. 2004 Nov;154 (21-22):523-7.

65. Weir MA et al. Micro-Particle Curcumin for the Treatment of Chronic Kidney Disease-: Study Protocol for a Multicenter Clinical Trial. Can J Kidney Health Dis. 2018 Dec 5;5: 2054358118813088.

66. 52. Kang DG et al. Butein ameliorates renal concentration ability in cisplatin-induced acute renal failure in rats. Biol Pharm Bull. 2004 Mar:27(3):336-70.

67. Brown AC. Kidney toxicity related to herbs and dietary supplements: Online table of case reports. Part 3 of 5 series. Food Chem Toxicol. 2017 Sep;107(Pt A):502-519.

68. Luciano RL and Perazella MA. Aristolochic acid nepthropathy: epidemiology, clinical presentation, and treatment. Drug Saf. 2015 Jan 38(1):55-64.

69. Fernando-Prado R. Nutrients Turned into Toxins: Microbiota Modulation of Nutrient Properties in Chronic Kidney Disease. Nutrients. 2017 May 12;9(5).

Disgusting Source of the Majority of Urinary Tract Infections

Disgusting Source of the Majority of Urinary Tract Infections

Urinary tract infections (UTIs) affect anywhere from 25%1 to 60%2,3 of women over the course of their lifetime. Research published in 2015, UTIs were responsible for 10.5 million doctor visits in the U.S. in 2007.4

A study5 published in the journal Open Forum Infectious Diseases in 2017 noted hospitalization rates for UTIs in the U.S. rose by 52% between 1998 and 2011 — a direct result of increasing antimicrobial resistance

According to this study, there were 400,000 UTI-related hospitalizations in 2011, with an estimated cost of $2.8 billion. The highest rates of increase were seen in women and older patients.

In the past, recurrent UTIs were thought to be caused by reinfection by the same pathogen,6 but recent research7,8,9 published in the Journal of Molecular Biology suggests this pattern has changed, and the reason why UTIs tend to have such a high recurrence rate in postmenopausal women is because the infection can be caused by several different pathogens.

According to the authors, the data uncovered via urine and bladder biopsies “suggest that diverse bacterial species and the adaptive immune response play important roles” in recurrent UTIs.

Pathogenic Mechanisms of UTIs

Women are more prone to urinary tract infections than men, in part because of their shorter urethras. Adult men have another factor going for them. The male prostate gland actually produces a bacterial growth inhibitor that is secreted directly into their urinary system.10

According to research11 published in 2015, several different pathogens can trigger a UTI; most commonly Escherichia coli (E.coli), Klebsiella pneumoniae, Proteus mirabilis, Enterococcus faecalis and Staphylococcus saprophyticus. Of these, about 80% to 90% are caused by E.coli,12,13 which is normally found in the intestinal tract.14

Problems only arise when this ordinary bacterium is present in high numbers in places where it shouldn’t be, like your urinary system. When E. coli gets into your urinary tract and multiplies, you experience the usual signs and symptoms of a UTI, such as:15

  • Burning with urination
  • Frequent urges to urinate
  • Lower abdominal pain or aching
  • Blood in your urine (sometimes, but not always)
  • Cloudy or foul-smelling urine

The reason your body cannot simply expel the E. coli through urination is because the bacteria are covered with tiny fingerlike projections called fimbria, made of an amino acid-sugar complex, a glycoprotein called lectin, which makes them sticky.

This stickiness allows the bacteria to adhere to the inner wall of your bladder and/or work their way upward toward your kidneys, at which point the situation can become quite serious.

Sepsis is another complication of untreated or unsuccessfully treated UTI (which can happen if the infection is caused by drug-resistant bacteria), which can be life-threatening. An infusion of intravenous vitamin C with hydrocortisone and thiamine has been shown to reduce mortality from sepsis nearly fivefold, but many health care professionals are still unaware of this revolutionary treatment.

In addition to the symptoms already mentioned, a UTI in an older individual can also result in sudden behavioral changes such as restlessness, agitation, lethargy or social withdrawal, mental confusion and even hallucinations and delirium.16

According to Dr. Amanda Smith, medical director at the Byrd Alzheimer’s Institute at the University of South Florida, symptoms of UTI in the elderly actually tend to be primarily behavioral,17 which can result in delayed diagnosis and treatment. So, doctors of elderly patients exhibiting these kinds of behavioral symptoms, especially when combined with low-grade fever, should have them checked for UTI.18

Recurrent UTIs Linked to Variety of Pathogens in Bladder Wall

What the Journal of Molecular Biology study discovered was those different types of bacteria form colonies deep in the tissue of the bladder wall, past the urothelium layer in many cases, making them very difficult to get rid of. As noted by Science Daily, which reported the Journal of Molecular Biology findings:19

“[F]or some postmenopausal women, UTIs recur so frequently that they become a chronic condition, requiring daily doses of increasingly powerful antibiotics as the infection-causing bacteria gradually become resistant to each new drug.

‘For older women, these infections can go on for tens of years,’ said Dr. Nicole De Nisco, assistant professor of biological sciences at UT Dallas and lead author of the study. ‘Eventually, a patient’s last resort might be removing the bladder’ …

To investigate the pathogenic mechanisms and immune responses related to recurring UTIs, De Nisco and her colleagues analyzed urine and biopsies from 14 postmenopausal women …

They found that in addition to the expected E. coli, bacteria in urine samples included Klebsiella pneumoniae and Enterococcus faecalis, while species in biopsied tissue included E. coli, Staphylococcus hominis and Bacillus firmus.

‘Our findings confirm that bacteria do form communities within the bladder wall of RUTI [recurrent UTI] patients, which was not previously known,’ De Nisco said. ‘This research is a critical step toward better understanding the mechanisms of recurring urinary tract infection and inflammation in postmenopausal women’ …

Future studies will focus on determining effective techniques to remove these bacteria and chronic inflammation from the bladder, finding new strategies to enhance immune system response, and pinpointing the various bacterial pathogens involved in RUTIs.”

Factory-farmed Chicken — The Leading Source of UTI Infections

Conventional wisdom has maintained UTIs are primarily caused by a transfer of naturally-occurring E. coli via sexual contact with an infected individual and/or the transfer of fecal bacteria from your anus to your urethra by poor personal hygiene. However, more recent studies have conclusively demonstrated that a majority of UTIs are actually caused by exposure to contaminated chicken.20

Importantly, factory-farmed chickens are the source of most antibiotic-resistant UTIs — a problem that can be traced back to the routine use of antibiotics for growth-promotion purposes, which has allowed resistance to develop. Drug-resistant E. coli strains from supermarket meat were matched to strains found in human E. coli infections as early as 2005.21

Research22,23 published in 2006 confirmed that humans could develop antibiotic resistance by eating poultry treated with antibiotics. Bacteria from conventional chicken, and those who ate such chicken, were found to be more prone to developing resistance against Synercid (generic names: quinupristin and dalfopristin24), a strong antibiotic used to treat vancomycin-resistant Enterococcus faecium.25

In essence, eating antibiotic-treated chicken can cause you to develop resistance to the last lines of defense currently available in the modern medicine cabinet — a steep price for inexpensive meat! As reported by Infectious Control Today:26

“Laboratory tests showed that the bacteria isolated from patients and vegetarians had no pre-existing resistance to Synercid. Resistance was rare among antibiotic-free poultry, but a majority of bacterial isolates from conventional poultry samples were resistant.

After exposure to virginiamycin, E. faecium from conventional poultry and from patients who consumed poultry became resistant to Synercid more often than E. faecium from vegetarians or from antibiotic-free poultry.

Some of the resistance was attributed to a specific gene, and both the gene and resistance were associated with touching raw poultry meat and frequent poultry consumption.”

Genetic Matching Links UTIs to Contaminated Chicken

American, Canadian and European studies27,28,29 published in 2012 all confirmed close genetic matches between drug-resistant E. coli collected from human patients and those found on poultry (chicken and turkey).

More recently, a study30 published in the journal mBio in 2018 found 79.8% of chicken, pork and turkey samples purchased from large retail stores in Flagstaff, Arizona, were contaminated with E. coli. The researchers also tested blood and urine samples from people who visited a major medical center in the area, finding E. coli in 72.4% of those diagnosed with a UTI.

In particular, a strain of E. coli known as E. coli ST131 showed up in both the meat samples (particularly poultry) and the human UTI samples. Most of the E. coli in the poultry was a variety known as ST131-H22, which is known to thrive in birds. This specific strain was also found in the human UTI samples.

“Our results suggest that one ST131 sub lineage — ST131-H22 — has become established in poultry populations around the world and that meat may serve as a vehicle for human exposure and infection,” the researchers noted, adding that this E. coli lineage is just one of many that may be transmitted from poultry and other meat sources to people.

Make Sure Your Chicken and Eggs Are Organic and Free-Range

While findings such as these are a potent reminder to use caution when handling raw chicken and to cook poultry thoroughly, another option — and perhaps the most sensible and rational approach is to avoid factory-farmed chicken altogether.

It’s easily among the most contaminated foods in the U.S., as a recent lawsuit against the U.S. Department of Agriculture for failing to address high rates of fecal bacteria on chicken can attest to. Factory-farmed chicken also has a weak nutritional profile compared to other protein sources, including pasture-raised chicken (which is also less likely to carry harmful contaminants).

For example, a study31,32,33 by the American Pastured Poultry Producers Association (APPPA), which compared the nutrient value of pastured chickens with the USDA’s National Nutrient Database for Standard Reference values for CAFO chicken, found pasture-raised chickens contained:

  • 406.8% more vitamin E (1.86 IUs per 100 grams compared to 0.367 IUs)
  • About half the fats of CAFO chicken (saturated, monounsaturated and polyunsaturated)
  • An average omega-3-to-6 ratio of 1-to-5, which is near ideal, compared to the USDA’s value of 1-to-1534

Considering the hazards associated with raw chicken, if you’re going to eat it, I recommend making sure it’s organic and free-range, pasture-raised. Ditto for eggs, as CAFO eggs are also far more prone to pathogenic contamination than organic pastured eggs.

Your best bet is to find a local source of organic, free-range eggs and chicken meat. The Cornucopia Institute’s egg report and scorecard ranks 136 egg producers according to 28 organic criteria, is an excellent resource if no local producers are available.

In June 2017, Cornucopia also began working on a chicken report and scorecard. Considering the egg report took six years to produce, it may still be a while before the chicken scorecard is ready. You can contribute to this report by following the simple instructions listed in their June 13 Action Alert.35

How to Treat a UTI at Home

As mentioned earlier, the fimbria (fingerlike projections) of E. coli are made of a sticky glycoprotein called lectin, which is why the bacteria are so hard to flush out. It’s not impossible however, even without an antibiotic. While antibiotics are typically the go-to treatment, you may be better off starting out with a D-mannose supplement.36

Mannose is produced by your cells and covers the internal lining of your urinary organs. The lectin on the bacteria’s fimbria binds to mannose, which is why the bacteria adhere to the walls of your urinary system.

When you take D-mannose, the E. coli adheres to the mannose present in your urine, which is then flushed out when you urinate. As the bacterial load on epithelial cells lessen, they’re more easily overtaken by agents of your immune system.

Infections caused by a bacterium other than E. coli may be eliminated by taking a saturated solution of potassium iodide (SSKI). Both of these treatments are recommended by Dr. Jonathan Wright, medical director of Tahoma Clinic in Tukwila, Washington, and the author of the book, “D-Mannose and Bladder Infection: The Natural Alternative to Antibiotics.”

For UTIs caused by bacteria or fungi other than E. coli, Wright suggests taking 15 drops of SSKI in water every three to four hours for two days (three days maximum).37 In order to know which of these treatments would work best, you’d need to perform a culture test to identify the bacteria responsible for your infection.

Alternatively, Wright suggests taking D-mannose first, and if significant improvement doesn’t occur, move on to SSKI. A culture test is also advisable to rule out a drug-resistant infection, as this will require close medical supervision to avoid serious complications.

UTI Prevention 101

Prevention is, of course, your best option, and as a woman, there are some specific hygiene steps you can take to maintain a healthy urinary tract:

Drink plenty of pure, filtered water every dayUrinate when you feel the need; don’t resist the urge to go
Wipe from front to back to prevent bacteria from entering your urethraTake showers instead of tub baths; avoid hot tubs/Jacuzzis
Cleanse male and female genital areas prior to sexual intercourse




Dr. Burgdorfer the discoverer of Lyme Disease for whom the spirochete was named after as Borellia Burgdorferi, recommended INVIVE Silver against Lyme Disease at a conference in Vancouver, Canada over 20 years ago

Over 500 M.D.s were at that conference, and Dr.Burgdorfer’s recommendations were and have been ignored to this day. The Doctors did “NOT” discount the MSP (Mild Silver Protein) Silver, but claimed/told Dr. Burgdorfer that they had better and more targeted drugs. To which Dr. Burgdorfer replied: Your supposed better and targeted drugs have resulted in/caused the pathogenic mutated nightmare world of today, because silver kills the pathogens instantly and completely, and silver does not allow the pathogens to mutate, while your drugs CAUSE mutations by allowing pathogens to survive though wounded, and thereby mutate and develop resistance to your drugs, causing drug resistant strains. E.g.: MRSA Methicillin-aminoglycoside resistant Staphylococcus aureus, MRSA An organism with multiple antibiotic resistances–E.g.: aminoglycosides, chloramphenicol, clindamycin, erythromycin, rifampin, tetracycline, streptomycin, cephalosporin.

We wouldn’t have this abx (antibiotic) resistant nightmare world, if the FDA had not taken away the silver from us when biased clerks at the FDA in 1938 took control of/took over our Food and Drug Supply.

NOTE: This Protocol must be followed exactly as written. Any deviation may result in a possible failure.

NOTE: For Chronic long-term Lyme Disease, this Protocol may have to be followed for 3-9 months. Definitely 3 months

NOTE: Invive Silver 5000 ppm is the only silver empirically proven to substantiate this protocol


  1. 5 cc per 30 lbs. body weight is the daily total dosage, and is the “target dosage”.
  2. There are 5 cc in a measuring teaspoon.
  3. A 120 lb. person would take 120 lbs. divided by 30 lbs.= 4 teaspoons per day
  4. It is taken in 4 divided doses. Which means 1 tsp. every 6 hours.
  5. You may use a measuring teaspoon which is 5 cc, (do not use unknown serving teaspoons).
  6. Inside the Silver Canister that the Silver Bottle arrives in, there is a Medical Graduated 1 ounce measuring cup for Medical Purposes graduated in cc and ml.
  7. Store the silver bottle in its canister always and keep it in a dark cupboard out of the light, because it is light sensitive like photographic film, and will be stressed by light.
  8. It can be boiled or frozen, BUT not exposed to light for over 1 minute when taken from its bottle.
  9. There are 118.291 cc/ml (round to 120 as extra is put in each bottle) = 4 oz. fl. U.S. in each bottle.
  10.  For a 120 lb. person take 4 teaspoons in total daily (per 24 hours) as a minimum dosage.
  11. Do NOT swallow. Hold silver sublingually under your tongue for 120 seconds. So that the silver will go into your bloodstream like an IV the same as a Nitroglycerin pill does to stop a heart attack.
  12. After 120 seconds: Gently swallow slowly the secretions of the mouth. After 120 seconds “slowly” swallow to allow further absorption via your esophagus. Do NOT expiate/spit out the silver. Therefore, swallow slowly any remaining after 120 seconds.
  13. Maintain a “Steady State” of Silver in your bloodstream by taking 5 cc every 6 hours, as this is necessary to kill the spirochetes every time they come out in the bloodstream from a sequestered area.
  14. We can reach the sequestered areas that the spirochetes hide in, due to coating with protein of the silver particle, with said coating slowly washing off like a time release, then releasing the silver ions, thus allowing this INVIVE silver to reach the sequestered areas of where the spirochetes are hiding. This coating provides a time release methodology for eradication of the spirochetes in sequestered areas.
  15. A Hot Bath for 40 minutes in the bathtub at 104 degrees, is mandatory.
  16. Purchase a $9 indoor/outdoor cord probe thermometer
  17. The hot 104-degree F. water expands the tiny capillary blood vessels, allowing silver to reach tissue areas where the Lyme spirochete is sequestered and hard to eradicate.
  18. Stay in tub for 40 minutes (without adding more hot water to heat up the tub) as this allows the water to cool down to 94 degrees F., so that you do not sweat when you get out of tub. Read a book, or meditate etc., to maximize benefit by your time in the tub.


  • 20 minutes “before” tub bath, imbibe/drink 4oz of citric juice, either orange or grapefruit juice —this allows the silver nano particles to cross over the blood brain barrier in case there is neurological involvement and inflammation which there usually is.
  • 1 minute before getting in tub, take a dosage of 5 cc of silver (1teaspoon)

Additional Facts:

 Must drink ½ of your body weight of Distilled Water in total DAILY, to prevent a “possible” Jarisch-Herxheimer reaction due to the release of toxins from the dying spirochetes.

  1.  All humans should imbibe/drink ½ their weight in ounces of water every day, even if they do not have Lyme Disease. We are all dehydrated and should be taught better.
  2. Treat until asymptomatic
  3. Then without pause, treat 1 more month to make sure that you have eradicated all the spirochetes.

Wholesale Cost of Silver 5000 ppm:

$539.94 (?) monthly per 6 pack of 4 oz. Bottles- 24 teaspoons in one 4 oz, bottle = 1 Bottle lasts 6 days